NOX介導(dǎo)的氧化應(yīng)激對肝纖維化相關(guān)信號通路調(diào)控的研究進展
發(fā)布時間:2018-09-11 10:13
【摘要】:肝纖維化是各種慢性肝臟疾病的共同病理結(jié)果,以細胞外基質(zhì)尤其是Ⅰ型和Ⅲ型膠原的過度沉積為主要特點,其持續(xù)進展可導(dǎo)致肝硬化,甚至肝癌。NADPH氧化酶(NOX)是一種多亞基組成的跨膜酶復(fù)合物,眾多研究表明其介導(dǎo)的氧化應(yīng)激在肝纖維化的發(fā)病機制中發(fā)揮重要作用,并參與調(diào)控多條肝纖維化相關(guān)信號通路,如TGF-β/Smad信號通路、MAPK信號通路、PI3K-AKT信號通路、NF-κB信號通路。
[Abstract]:Hepatic fibrosis is a common pathological result of various chronic liver diseases. It is characterized by excessive deposition of extracellular matrix, especially type I and type 鈪,
本文編號:2236395
[Abstract]:Hepatic fibrosis is a common pathological result of various chronic liver diseases. It is characterized by excessive deposition of extracellular matrix, especially type I and type 鈪,
本文編號:2236395
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